Parkinson disease is a neurodegenerative disorder that affects millions of people globally. It is caused by the loss of dopamine-producing neurons in the brain, resulting in motor and non-motor symptoms that can significantly affect individuals quality of life. While current treatments aim to alleviate the symptoms of Parkinsons disease, ongoing research into its neural basis holds promise for developing more effective therapies and addressing ethical concerns.
The neural basis of acute pain is a complex process that involves the nervous system, the brain, and the limbic system. Research on the neural basis of pain has potential applications in the development of effective treatments and therapies for pain management. However, there are challenges and limitations associated with this research.
Chronic pain affects approximately 20% of the global population, and its management can be challenging due to the complex mechanisms underlying its development. In this blog post, we delve into the neural basis of chronic pain, including the role of peripheral and central sensitization, neural plasticity, and descending modulation. We also explore potential applications of the research in developing new therapies and technologies for pain relief, and highlight the challenges and limitations that still exist in our understanding of chronic pain.