Neural basis of dyslexia
This article explores the neural basis of dyslexia, including its prevalence, causes, and symptoms. We review the current state of research related to this disorder, highlighting the importance of understanding neurobiological underpinnings for the development of effective interventions. Future research scopes and challenges are discussed as well.
Dyslexia is a neurodevelopmental disorder that affects approximately 5-10% of the population. Despite adequate intelligence, educational opportunities, and motivation, individuals with dyslexia have difficulty with phonological processing, decoding words, reading fluency, spelling, comprehension, and writing. Recent advances in neuroscientific research have shed light on the neural mechanisms that underlie this disorder.
Research has shown that several brain regions are involved in reading and language processing, including the left occipitotemporal cortex, left angular gyrus, and left inferior frontal gyrus. Individuals with dyslexia have less activation of these regions, suggesting they experience difficulty recognizing and manipulating individual sounds in words, processing visual information, and integrating it into language comprehension.
The genetic basis of dyslexia has also been studied, and researchers have identified genetic variations in several genes related to brain development and function that contribute to dyslexia. Diagnosing dyslexia involves a comprehensive assessment of reading and language skills, cognitive ability, and academic history. Although currently no medication can cure dyslexia, evidence-based treatments, including phonics-based reading instruction and specialized reading programs, can improve reading skills.
Future research may involve identifying specific subtypes of dyslexia in terms of different underlying causes, developing personalized interventions using neurofeedback to train specific neural pathways, and studying the benefits of multisensory approaches in an educational setting.